주메뉴 바로가기 본문내용 바로가기 사이트정보 바로가기

논문특허 2차년도

제목 - 설명
  • [논문] Structural basis for the interaction between human Npl4 and Npl4-binding motif in human Ufd1

    • 등록일
      2023.02.06
    • 조회수
      191

•연구자: 화학과 강원철

 

•발표일: 2022.11

 

•DOI:https://doi.org/10.1016/j.str.2022.08.005

 

•Thang Quyet Nguyen et al., Structure (Q1); 30(11), Pages 1530-1537.e3, 2022

 

•Abstract
The heterodimer of human ubiquitin fusion degradation 1 (hUfd1) and human nuclear protein localization 4 (hNpl4) is a major cofactor of human p97 adenosine triphosphatase (ATPase). The p97-Ufd1-Npl4 complex translocates the ubiquitin-conjugated proteins from the endoplasmic reticulum membrane to the cytoplasm. Ubiquitinated proteins are then degraded by the proteasome. The structures of Npl4 and Ufd1-Npl4 (UN) complex in Saccharomyces cerevisiae have been recently reported; however, the structures of hNpl4 and the human UN complex remain unknown. Here, we report the crystal structures of the human UN complex at a resolution of 2.7 Å and hNpl4 at a resolution of 3.0 Å. We also present atomic details and characterization of the human UN complex. Crystallographic studies and site-directed mutagenesis of the hUfd1 residues involved in the interaction with hNpl4 revealed the atomic details of the two proteins.

상단으로 이동